BTN3A1 promotes tumor progression and radiation resistance in esophageal squamous cell carcinoma by regulating ULK1-mediated autophagy
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Research suggests that a specific factor, BTN3A1, may contribute to tumor progression and radiation resistance in esophageal squamous cell carcinoma by regulating a particular cellular process. The relationship between molecular markers and radiotherapy efficacy is also being explored in other types of cancer, such as glioma. However, the specific mechanisms and factors involved in radiation resistance and tumor progression may vary across different cancer types.
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