Research suggests that the SARS-CoV-2 Nsp6 protein may damage the hearts of drosophila and mouse cardiomyocytes through increased glycolysis mediated by the MGA/MAX complex. However, the extent of this damage may vary among different variants of the protein, with the omicron variant potentially causing less harm. The study of SARS-CoV-2 proteins and their effects on cardiovascular health is ongoing, with some investigations exploring potential therapeutic targets.