Restoration of ER proteostasis attenuates remote apoptotic cell death after spinal cord injury by reducing autophagosome overload | Cell Death & Disease
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Research suggests that restoring endoplasmic reticulum proteostasis may help reduce cell death after spinal cord injury. This is achieved by decreasing the overload of autophagosomes, which are involved in cell recycling. The findings indicate a potential link between endoplasmic reticulum function and the mitigation of remote apoptotic cell death following spinal cord injury.