Research suggests that NLRC5 plays a role in promoting tumorigenesis in cervical cancer by regulating a specific signaling pathway. The exact mechanisms and pathways involved are still being studied, with some findings indicating a connection to the PI3K/AKT signaling pathway. Other studies are exploring various factors and modifications that may contribute to cancer progression, including protein ubiquitination, arginine methylation, and non-coding RNAs, although their direct relation to NLRC5 and cervical cancer is unclear.